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Effects of diazoxide on Fas/FasL protein expressions in rat myocardium suffered from long-term hypothermic preservation

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Author:: FAN Ying()
ZHENG Ming-Zhi()
GUO Wei()
JIANG Jian-Ping()
ZHU Li()
SHEN Yue-Liang()
CHEN Ying-Ying()

Journal Title:: ACTA PHYSIOLOGICA SINICA

Issue:: Volume 60, Issue 01, 2008

DOI:

Key Word:

Abstract：

Reference

[1]Wheeldon D.Thoracic organ preservation.Perfusion 1991;6(3):191-202.
[2]XuM(徐曼),LiQF,ZhangWH,ZhangHQ,XuCQ,Wang XM.Effect of Dan Shen Pill on expression of Fas/FasL proteins in cultured neonatal rat cardiomyocytes during hypoxia and reoxygenation.Chin J Pathophysiol(中国病理生理杂志)2003;19(4):499-502.(Chinese,English abstract)
[3]Akao M,Ohler A,O'Rourke B,Marban E.Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells.Circ Res 2001;88(12):1267-1275.
[4]Wakiyama H,Cowan DB,Toyoda Y,Federman M,Levitsky S,MaCully JD.Selective opening of mitochondrial ATPsensitive potassium channels during surgically induced myocardial ischemia decreases necrosis and apoptosis.Eur J Cardiothorac Surg 2002;21(3):424-433.
[5]Guo W(郭炜),Shen YL,Chen YY,Hu ZB,Yah ZK,Xia Q.The protective effect of diazoxide on long-term heart preservation.Acta Physiol Sin(生理学报)2004;56(5):632-638(Chinese,English abstract).
[6]Babcock DF,Herrinqton J,Goodwin PC,ParK YB,Hille B.Mitochondrial participation in the intracellular Ca2+ network.J Cell Biol 1997;136(4):833-844.
[7]Chen YY(陈莹莹),Shen YL,Cao CM,Xu WH,Qian ZM,XiaQ.Hydrogen peroxide augments the injury effect of iron on the isolated rat heart and cardiomyocytes.Acta Physiol Sin (生理学报)2001;53(3):175-182(Chinese,English abstract).
[8]Das DK,Maulik N.Conversion of death signal into survival signal by redox signaling.Biochemistry(Mosc)2004;69(1):10-17.
[9]HartH,LongH,WangH,WangJ,ZhangY,WangZ.Progressive apoptotic cell death triggered by transient oxidative insult in H9c2 rat ventricular cells:a novel paRem of apoptosis and the mechanisms.Am J Physiol Heart Circ Physiol 2004;286(6):2169-2182.
[10]Griffiths EJ,Halestrap AP.Mitochondrial non-specific pores remain closed during cardiac ischaemia,but open upon reperfusion.Biochem J 1995;307(Pt 1):93-98.
[11]Hausenloy DJ,Maddock HL,Baxter GF,Yellon DM.Lnhibiting mitochondrial permeability transition pore opening:a new paradigm for myocardial precOnditiOning?Cardiovasc Res 2002;55(3):534-543.
[12]Ozcan C,Bienenqraeber M,Dzeja PP,Terzic A.Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.Am J Physiol Heart Circ Physiol 2002;282(2):531-539.
[13]Akao M,O'Rourke B,Kusuoka H,Teshima Y,Jones S P,Marban E.Differential actions of cardioprotective agents on the mitochondrial death pathway.Circ Res 2003;92(2):195-202.
[14]Kaufmann SH,Earnshaw WC.Induction of apoptosis by cancer chemotherapy.Exp Cell Res 2000;256(1):42-49.
[15]Condo I.Testi R.Intracellular mediators of programmed cell death initiated at the cell surface receptor Fas.Transpl Int 2000;13(Suppl 1):S3-S6.
[16]Oshimi Y,Miyazaki S.Fas antigen-mediated DNA fragmentation and apoptotic morphologic changes are regulated by elevated cytosolic Ca2+level.J Immtmol 1995;154(2):599-609.
[17]Oh SH.Lim SC.A rapid and transient ROS generation by cadmium triggers apoptosis via caspase-dependent pathway in HepG2 cells and this is inhibited through N-acetylcysteinemediated catalase upregulation.Toxicol Appl Pharmacol 2006;212(3):212-223.
[18]Provinciali M,Donnini A,Arqentati K,Di Stasio G.Bartozzi B.Bernardini G.Reactive oxygen species modulate Zn2+induced apoptosis in cancer cells.Free Radic Biol Med 2002:32(5):431-445.
[19]Tirosh O,Aronis A,Melendez JA.Mitochondrial state 3 to 4 respiration transition during Fas-·mediated apoptosis controis cellular redox balance and rate of eell death.Biochem Pharmacol 2003;66(8):1331-1334.
[20]Sutuki Y,Ono Y.Hirabayashi Y.Rapid and specific reactive oxygen species generation via NADPH oxidase activation during Fas-mediated apoptosis.FEBS Lett 1998;425(2):209-212.