ROLE OF CYTOSOLIC CALCIUM BALANCE ON CELL INJURY IN CULTURED BOVINE AORTIC ENDOTHELIAL CELLS DURING HYPOXIA AND REOXYGENATION

( views:, downloads: )
Author:
CHEN LING-HONG()
ZHOU Zhao-Nian()
Journal Title:
ACTA PHYSIOLOGICA SINICA
Issue:
Volume 49, Issue 03, 1997
DOI:
Key Word:

Abstract:

  • [1]Janssens SP, Shimouchi A, Quertermous T, et al. Cloning and expression of a cDNA encoding human endothelial-derived relaxing factor/nitric oxide synthases. J Biol Chem, 1992, 2 6 7 : 14519 ~ 14522
  • [2]Buckley BJ, Barchowsky A, Dolor RJ, et al. Regulation of arachidonic acid release in vascular endothelium.Ca2+-dependent and-independent pathways. Biochem J, 1991, 280 : 281 ~ 287
  • [3]Madden MC, Vender RL, Friendman M. Effect of hypoxia on prostacyclin production in cultured pulmonary artery endothelium. Prostaglandins, 1986, 31: 1049~1062
  • [4]Channon JY, Ledie CC. A calcium-dependent mechanism for associating a soluble arachidonoyl-hydrolyzing phospholipase A2 with membrane in the macrophage cell line. J Biol Chem, 1990, 265:5409~5413
  • [5]Clark JD, Lin LL, Kriz RW, et al. A novel arachidonic acid-selective cytosolic PLA2 contains a Ca2+-dependent translocation domain with hymology to PKC and GAP. Cell, 1991, 65: 1043 ~ 1051
  • [6]Sunnergen KP, Rovetto MJ. Myocyte and endothelial injury with ischemia reperfusion in isolated rat hearts.Am J Physiol, 1987, 252: H1211~H1217
  • [7]Lindal S, Sorlie D, Jorgensen L. Endothelial cells of the cardiac microvasculature during and after cold cardioplegic ischemia. Scand J Thor Cardiovasc Surg, 1988, 22: 257~265
  • [8]Arnould T, Michiels C, Alexandre I, et al. Effect of hypoxia upon intracellular calcium concentration of human endothelial cells. J Cellular Physiol, 1992, 152:215~222
  • [9]Stevens T, Cornfield DN, Mcmurtry IF, et al. Acute reductions in PO2 depolarize pulmonary artery endothelial cells and decrease [Ca2+]. Am J Physiol, 1994, 266:H1416~H1421
  • [10]Schilling WP, Rajan L, Strobl-Jager E. Bradykinin-stimulated calcium influx in cultured bovine aortic endothelial cells. Am J Physiol, 1988, 255:H219~H227
  • [11]Post JM, Hume JR, Archer SL, et al. Direct role for potasium channel inhibition in hypoxic pulmonary vasoconstriction. Am J Physiol, 1992, 262:C882~C890
  • [12]Lopez-Barneo J, Lopez-Lopez JR, Urena J, et al. Chemotransduction in the carotid body: K current modulated by PO2 in the type I chemopreceptor cells. Sci Wash DC, 1988, 241
  • [13]Lopez-Lopez J, Gonzaez C, Urena J, et al. Low PO2 selectively inhibits K channel activity in chemoreceptor cells of the mammalian carotid body. Physiol, 93 : 1001 ~ 1015
  • [14]Demirel E, Laqskey RE, Purkerson S, et al. The passive calcium leak in cultured porcine aortic endothelial cells. Biochem Biophys Res Commun, 1993, 3:1197~1203
  • [15]Revest PA,Abbott JN. Membrane ion channels in endothelial cells. Trends Pharmacol Sci, 1992,13:404~407
  • [16]Clague JR, Langer GA. The pathogenesis of free radical-induced calcium leak in cultured rat cardiomyocytes.J Mol Cell Cardiol, 1994, 26: 11~21
  • [17]Horgan MJ, Lum H, Malik AB. Pulmonary edema after pulmonary artery occlusion and reperfusion. Am Rev Respir Dis, 1989, 140:1421~1428
WanfangData CO.,Ltd All Rights Reserved
About WanfangData | Contact US
Healthcare Department, Fuxing Road NO.15, Haidian District Beijing, 100038 P.R.China
Tel:+86-010-58882616 Fax:+86-010-58882615 Email:yiyao@wanfangdata.com.cn